1.   Increased basal acid output and increased peak acid output are important features in patients with DU.

2.   We also found the basal acid output to be significantly greater in patients with duodenal ulcers and with H pylori than in patients with duodenal ulcers without this infection.

3.   The median basal acid output was similar in the H pylori negative and positive healthy volunteers.

4.   The basal acid output in the one duodenal ulcer subject in whom H pylori infection was not eradicated was similar before and after the triple treatment.

5.   Though gastrin is considered to be the main mediator of food stimulated acid secretion, its role in the regulation of basal acid output is unclear.