61.   Prostaglandins inhibit acid secretion and their synthesis and release by gastric mucosal cells is enhanced by exposure to ethanol.

62.   Substances with high osmolarity have been found to inhibit acid secretion.

63.   Alcoholic beverages with low ethanol content stimulate gastric acid secretion by additional mechanisms.

64.   None of the known stimulants of acid secretion present in beer given alone or in combination could be implicated.

65.   These preliminary results suggest that the powerful stimulants of gastric acid secretion are thermostable and anionic polar substances.

66.   Cyclical changes in H pylori infection may cause the variations in basal acid secretion that are seen in duodenal ulcer disease.

67.   Despite this reduction in plasma gastrin, no consistent changes in rates of acid secretion have been documented after eradication treatment.

68.   Gastric acid secretion was measured by aspiration as described by Baron.

69.   The largest dose produces maximal stimulation of acid secretion.

70.   The rate of acid secretion was calculated with correction for pyloric losses and duodenogastric reflux by phenol red recovery and sodium measurement respectively.