1.   Blockade of potassium channels or augmentation of the calcium current can produce EADs that lengthen repolarisation and cause triggered activity and ventricular tachyarrhythmias experimentally.

2.   Er one potassium channel which is commonly worked on is known as the delayed rectifier.

3.   And the repolarization is associated here with the opening of potassium channels.

4.   There are no very highly specific organic inhibitors for potassium channels.

5.   Evolution seems to have er devised a large number of specific inhibitors for sodium channels but not for potassium channels.

6.   And you can also determine from the sequence that the common evolutionary origin er was er centred around potassium channels.

7.   The normal role of the new genes is to make critical components of nerve cells known as voltage-gated potassium channels.