61.   The gene studied by the Italian team makes a protein that triggers the self-destruct process in response to oxygen damage.

62.   The gene that makes the protein is inserted in the virus.

63.   The faulty gene either fails to make its protein or makes a different one, causing a characteristic change in the function of the affected cells.

64.   The gene normally makes a protein called fibrillin, which forms hairlike structures called microfibrils that give ligaments, tendons, artery walls and other tissues strength and resilience.

65.   The knockout mice have high levels of a cellular protein called Fos B, which means that the gene that makes this protein is highly active.

66.   The nucleic acid DNA stores genetic information, which is then copied into RNA and used to make proteins.

67.   The normal version of this gene directs nerve cells to make neurofilament proteins that travel from the globular cell body into the long axons that project from it.

68.   The products are referred to as Bt, but the bacteria themselves remain at the factory, making more proteins.

69.   The researchers bred mice without the gene that makes the protein.

70.   The researchers discovered that it directs cells to make a protein that is in membranes and helps move nutrients into cells.