21.   The relationship between H pylori, age, and gastric acid secretion is important as H pylori infection causes active chronic gastritis which may progress to gastric atrophy.

22.   All infected subjects had active chronic gastritis on histological examination.

23.   The prevalence of active chronic gastritis and subsequent gastric atrophy increases with age.

24.   After eradication of H pylori, duodenal ulcers do not usually recur and the associated chronic gastritis gradually disappears.

25.   Histological examination confirmed a severe chronic atrophic gastritis.

26.   Histological examination of these areas at this time confirmed a chronic gastritis and atrophic gastric mucosa.

27.   Chronic atrophic gastritis and chronic active gastritis were graded as mild, moderate and severe.

28.   In both series, H pylori was associated with chronic active gastritis compared with its presence in the mucosa without active inflammation.

29.   In contrast, antral tumours mainly arose from mucosa showing the changes of chronic active gastritis.

30.   The strong association between antral tumours and chronic active gastritis suggests the possibility that H pylori infection may have a pathogenic role.