1.   The finding of enhanced fasting gastrin concentrations in H pylori positive subjects and in duodenal ulcer disease can not easily be explained.

2.   Most investigators have observed that H pylori infection causes a greater percentage increase in the postprandial gastrin than fasting gastrin.

3.   The results clearly indicate that fasting gastrin and pepsinogen-I and -II concentrations were significantly higher in H pylori positive compated with H pylori negative subjects.

4.   The finding of enhanced fasting gastrin concentrations in H pylori positive subjects and in duodenal ulcer disease cannot easily be explained.