81.   What is needed is an ulcer model which is a single, truly chronic, proximal duodenal ulcer.

82.   Hyperpepsinogenaemia A has been shown in patients with duodenal ulcer disease, Zollinger-Ellison syndrome, hypertrophic gastropathy, chronic renal failure, and during omeprazole treatment.

83.   Hyperpepsinogenaemia A has often been seen in duodenal ulcer, Zollinger-Ellison syndrome, hypertrophic gastraophy, and chronic renal failure.

84.   High values of serum pepsinogen C were frequently found in all groups of patients with hyperpepsinogenaemia A.

85.   The high pepsinogen C concentrations in patients with duodenal ulcer are probably related to H pylori related chronic active gastritis.

86.   It has also been shown recently that, in patients with untreated duodenal ulcer, argyrophil cells were significantly more numerous in women than in men.

87.   Lysolecithin was the smallest component in the duodenal ulcer and chronic atrophic gastritis groups.

88.   The phosphatidylethanolamine value was higher in duodenal ulcer and lower in chronic atrophic gastritis compared with the control group.

89.   In chronic atrophic gastritis there was an appreciable amount of phosphatidylglycerol not present in patients with duodenal ulcer or in the control group.

90.   There was no significant difference in phospholipid composition between antral and duodenal sites in duodenal ulcer patients.