31.   An initial diagnosis of duodenal ulcer and the method of diagnosis being a radiographic examination seems to increase the risk of becoming a longterm user of ulcer drugs.

32.   Although duodenal ulcers rarely form in patients with achlorhydria, the widely held belief that all patients with duodenal ulcer disease secrete excessive amounts of acid is incorrect.

33.   The aim of our study was to find out if the more potent prokinetic drug cisapride could prevent duodenal ulcer relapse.

34.   Treatment failure was defined as evidence by endoscopy of a recurrent duodenal ulcer crater.

35.   All carriers of the bacterium, however, do not suffer from duodenal ulcer.

36.   Numerous studies have shown that Helicobacter pylori infection of the gastric antrum raises circulating gastrin concentrations in duodenal ulcer patients and healthy volunteers.

37.   The role of the increased serum gastrin concentration induced by H pylori in the pathogenesis of duodenal ulcer disease is also unknown.

38.   We have show, however, that there is no change in parietal cell sensitivity to gastrin after eradication of H pylori in duodenal ulcer patients.

39.   Risk factors for delayed duodenal ulcer healing are mentioned in all textbooks as important considerations in the management of duodenal ulcer disease.

40.   The number of duodenal ulcer episodes that had occurred during the previous two years was noted.