31.   Our findings may also have clinical implications concerning longterm treatment of acid related disorders with potent inhibitors of acid secretion.

32.   In healthy subjects hyperglycaemia induced by intravenous infusion of glucose reduces gastric emptying of solid and liduid meals, gastric acid secretion, and pancreatic secretion.

33.   During an acute hyperglycaemia induced by intravenous infusion of glucose, gastric emptying, gastric acid secretion and pancreatic enzyme secretion are reduced in healthy subjects.

34.   The fact that pentagastrin increased platelet activating factor precursors would suggest that platelet activating factor may have a role in the gastric functions, in particular gastric acid secretion.

35.   Our study shows that bFGF, even at a dose that did not affect gastric acid secretion, was also effective in healing these lesions.

36.   Obviously, this healing effect could not be mediated by the inhibition of acid secretion and other mechanisms are involved in the healing process.

37.   Studies to date have produced conflicting evidence concerning changes in acid secretion after eradication of H pylori and lowering of the serum gastrin concentration.

38.   The lack of fall in acid secretion despite a considerable fall in biologically active gastrin could be explained by H pylori altering the parietal cell sensitivity to gastrin.

39.   The failure of studies to show this clearly is probably related to the technical difficulties involved in measuring meal stimulated acid secretion.

40.   Pure ethanol in low concentrations is a mild stimulant of acid secretion whereas at higher concentrations it has either no effect or a mildly inhibitory one.