31.   The median basal acid output was similar in the H pylori negative and positive healthy volunteers.

32.   The basal acid output in the one duodenal ulcer subject in whom H pylori infection was not eradicated was similar before and after the triple treatment.

33.   This study shows that chronic H pylori infection is accompanied by appreciably increased gastric acid output in both healthy volunteers and duodenal ulcer patients.

34.   Though gastrin is considered to be the main mediator of food stimulated acid secretion, its role in the regulation of basal acid output is unclear.

35.   In our studies pepsin output correlated closely with acid output.