1.   Discussion Our present studies implicate active oxygen and lipid peroxidation in the pathogenesis of gastric mucosal injury induced by indomethacin.

2.   We have examined the hypothesis that the release of tissue type plasminogen activator may play a prominent role in endothelin induced gastric mucosal injury.

3.   These results give rise to the hypothesis that endothelin might increase the tissue type plasminogen activator activity accompanying the gastric mucosal injury.

4.   The purpose of this study was to determine by endoscopic examination the prevalence of oesophageal mucosal injury in asthmatics.

5.   Platelet activating factor causes gastrointestinal mucosal injury and ischemic bowel necrosis.

6.   Platelet activating factor plays a part in the mucosal injury of the gastrointestinal tract in anomals.