71.   Although not showing these features, the colonic and rectal biopsies from our patient showed changes unlike those usually seen in idiopathic inflammatory bowel disease.

72.   Nevertheless the absence of recurrence during long term follow up will be required to exclude underlying idiopathic inflammatory bowel disease.

73.   Patients with familial polyposis coli, inflammatory bowel disease, malabsorption syndrome, or a personal history of colonic resection were excluded.

74.   All subjects had a normal diet and were free of inflammatory bowel disease and of benign and malignant tumours at the time of complete colonoscopy.

75.   The cause of RP may be linked to the still unelucidated immunological mechanisms in inflammatory bowel disease that are clinically manifested as extraintestinal complications.

76.   Reports of in vitro production of cytokines by peripheral or mucosal mononuclear cells in inflammatory bowel disease, however, conflict.

77.   Normal colonic tissue and tissues from inflammatory bowel disease and colorectal tumours were obtained from the department of surgery at our university.

78.   Serum samples were obtained from healthy blood donors, patients with metastatic colorectal carcinoma without evidence of infection, and from patients with inflammatory bowel disease.

79.   This proctitis is a gluten driven phenomenon, therefore, and not the result of idiopathic inflammation or, in other words, distal inflammatory bowel disease.

80.   Until the aetiology of inflammatory bowel disease is established the development of new forms of treatment is limited.